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Pet Cats Cancer Genes Study Shows Link to Human Cancer

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Pet Cats Cancer Genes Study Shows Link to Human Cancer

A new study shows that pet cats’ cancer genes closely match those found in humans. Researchers say this discovery could improve cancer treatment for both species. Cancer behaves in similar ways in cats and people, which makes this link important.
Scientists examined around 500 pet cats from several countries. They used tissue samples already collected for veterinary care. In addition, they studied nearly 1,000 cancer-related genes to compare patterns.

Shared Genetic Mutations in Cats

The research identified 31 important driver genes linked to cancer. For example, the TP53 gene appeared in many feline tumors. This gene also plays a major role in human cancers. These mutations can stop the body from controlling tumor growth. As a result, cancer spreads more easily. Therefore, understanding these changes can guide better treatments. Experts from the Wellcome Sanger Institute say this overlap is significant. It shows how closely animal and human health connect.

Environmental Risks and Research Value

Pet cats live in the same environment as their owners. Therefore, they face similar risks like pollution and chemicals. This shared exposure adds value to research. Cancer is one of the top causes of illness in cats. However, scientists still know little about how it develops. As a result, this study fills a key research gap.
According to the European Pet Industry Federation, many households own cats. This helps researchers access more data.

Breast Cancer Similarities

The study also found strong links in breast cancer. Feline mammary cancer closely matches human breast cancer. Both share genes that control cell growth. For instance, the FBXW7 gene changed in over half of cat tumors. In humans, this gene links to more severe cases. Therefore, studying cats may improve treatment options. Researchers from the University of Bern believe this work can guide future therapies. It may benefit both humans and animals.

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